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科研方向：帕金森病综合征的基础和临床研究

发表文章：

第一作者文章： 1. SH-SY5Y human neuroblastoma cell line: in vitro cell model of dopaminergic neurons in Parkinson’s disease. Chin Med J, 2010,123(8):1086-1092. 2. Proteomics analysis of MPP+-induced apoptosis in SH-SY5Y cells. Neurol Sci, 2011,32:221–228. 3、Hongrong Xie, Hui Hu, Ming Chang, Dongya Huang, Xiaobo Gu, Xinli Xiong, Ran Xiong, Linsen Hu, Gang Li. Identification of chaperones in a MPP+-induced and ATRA/TPA-differentiated SH-SY5Y cell PD model. Am J Transl Res 2016;8(12):5659-5671. 4. 可溶性耐药相关钙结合蛋白在MPP+诱导的SH-SY5Y细胞PD模型中的表达及意义.吉林大学学报(医学版), 2011,37(4):626-630. 5. MPTP/MPP+诱导的PD实验模型的研究进展. 医学综述, 2011,17(5):669-671. 6. 蛋白质组学在中枢神经系统退行性疾病的研究进展.医学综述, 2011,17(6):828-830. 7. 应用两种方法诱导SH-SY5Y细胞分化的研究. 现代生物医学进展, 2011,11(1):63-67. 8. 不同分化状态的SH-SY5Y细胞对MPP+的敏感性. 中国老年学杂志, 2012,19(32):4224-4227. 9. 卒中及短暂性脑缺血发作二级预防指南(第五部分). 中国卒中杂志, 2014,9(12):1024-1040. 合作者文章： 1. Two potentially specific but relevant patterns of proteomic change. Neural Regen Res, 2010,5(20):1525-1533. 2. Proteomics analysis of methylglyoxal-induced neurotoxic effects in SH-SY5Y cells. Cell Biochem Funct, 2011,29:30-35. 3. Alpha-tubulin deacetylase as a potential and novel target for the prevention of Parkinson’s disease progression. Neural Regen Res, 2011,6(2):131-135. 4. HDAC6 α-tubulin deacetylase: A potential therapeutic target in neurodegenerative diseases, Neurol Sci Epub,2011,304:1-8. 5. 6-羟基多巴胺诱导SH-SY5Y细胞PD模型中peroxiredoxin6表达的研究. 中风与神经疾病杂志, 2009,26(6):668-670. 6. 6-羟基多巴胺诱导SH-SY5Y细胞PD模型中14-3-3蛋白表达的研究.中国老年学杂志, 2010,30(5):662-664. 7.多巴胺诱导SH-SY5Y细胞PD模型中GRP78的表达. 吉林大学医学版, 2010,36(1):76-80. 8. MPTP/MPP+诱导神经元凋亡的机制研究. 现代生物医学进展, 2011,11(5):964-967.